Der Endothelinrezeptorantagonist Macitentan verbessert die Isosorbid-5-Mononitrat (ISMN) und Isosorbiddinitrat (ISDN)-induzierte endotheliale Dysfunktion, oxidativen Stress und vaskuläre Inflammation

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dc.contributor.authorRoohani, Siyer
dc.date.accessioned2021-01-06T07:37:43Z
dc.date.available2021-01-06T07:37:43Z
dc.date.issued2021
dc.identifier.doihttp://doi.org/10.25358/openscience-5504
dc.identifier.urihttps://openscience.ub.uni-mainz.de/handle/20.500.12030/5508
dc.identifier.urnurn:nbn:de:hebis:77-openscience-883688bf-418f-4088-871c-60b82b10c86c3
dc.language.isogerde
dc.rightsInC-1.0*
dc.rights.urihttps://rightsstatements.org/vocab/InC/1.0/*
dc.subject.ddc610 Medizinde_DE
dc.subject.ddc610 Medical sciencesen_GB
dc.titleDer Endothelinrezeptorantagonist Macitentan verbessert die Isosorbid-5-Mononitrat (ISMN) und Isosorbiddinitrat (ISDN)-induzierte endotheliale Dysfunktion, oxidativen Stress und vaskuläre Inflammationde_DE
dc.typeDissertationde
jgu.date.accepted2020-11-27
jgu.description.extentVI, 131 Blätterde
jgu.organisation.departmentFB 04 Medizinde
jgu.organisation.nameJohannes Gutenberg-Universität Mainz
jgu.organisation.number2700
jgu.organisation.placeMainz
jgu.organisation.rorhttps://ror.org/023b0x485
jgu.rights.accessrightsopenAccess
jgu.subject.ddccode610de
jgu.type.dinitypePhDThesisen_GB
jgu.type.resourceTextde
jgu.type.versionOriginal workde

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